hypertrophy of heart
英 [haɪˈpɜːtrəfi ɒv hɑːt]
美 [haɪˈpɜːrtrəfi əv hɑːrt]
网络 心脏肥大; 心肥大
双语例句
- Electrocardiogram expression is right ventricle hypertrophy, certain guide wave of lobar sex P is seen on couplet, heart report axis is right slant.
心电图表现为右心室肥大，某些导联上可见肺性p波，心电轴右偏。 - The results indicated that GLXBBXD had the function of reducing the pulmonary hypertension and reversing the hypertrophy of right heart obviously.
结果表明，本制剂有明显的降肺动脉高压、右心室压和逆转右心肥大的作用。 - Long-term treatment with endothelin-receptor antagonist has improved haemodynamics, decreased left ventricular hypertrophy, increased cardiac output, and prolonged the survival rate of patients with chronic heart failure.
长期应用内皮素受体拮抗剂可改善心力衰竭患者的血流动力学、减缓左室重构、增加心脏指数、同时提高生存率。 - Hypertrophy of left ventricle worsens the function of right heart.
左室肥厚加重右心功能的损害。 - Pathological cardiac hypertrophy is one of adaptive responses of the heart to a variety of pathological stimuli, and always one of the complications of some cardiovascular diseases, such as hypertension, myocardial infarction, congenital heart diseases, cardiovalvular diseases, and etc.
病理性心肌肥厚是高血压、心脏瓣膜病、急性心肌梗塞及先天性心脏病等临床常见疾病的一种并发症，是心脏对多种病理刺激的一种适应性反应。 - Left ventricular hypertrophy was determined according to the traverse diameter of myocyte ( TDM) and by the ratio of heart weight to body weight ( HW/ BW).
用心肌细胞横径（TDM）和心脏湿重／体重（HW／BW）判定左心室肥厚。 - Clinical study on the Reversion Effect of Sini Decoction on left ventricle hypertrophy of patient with coronary heart disease
四逆汤对冠心病患者左心室肥厚逆转作用的临床研究 - [ Background] Left ventricular hypertrophy ( LVH) is one of the most common complications of hypertension, which is not only the results of compensated cardiac tissue growth due to heart overload, but also the pathological process of heart remodeling.
[研究背景]心肌肥大是高血压病最常见的并发症之一，它既是高血压病心脏负荷过大引起心肌组织代偿性生长的结果，也是心脏的病理性重构过程。 - The important pathophysiology process of left ventricular remodeling after AMI including myocyte hypertrophy and increase of collagen etc, which are main reason of left ventricular dysfunction and heart failure.
AMI后发生左室重构的主要病理生理变化包括心肌肥厚，心肌成纤维细胞异常增生及胶原合成增加等，是引起AMI后左室功能障碍，导致心力衰竭的主要原因。 - To study the significance of TGF β 1 expression in heart hypertrophy. The Sirius Red and immunohistochemical staining were used to detect the changes of cardiac fibrosis and TGF β 1 expression in heart and cardiocytes hypertrophy.
为探讨人心肌肥大时转化生长因子β1（TGFβ1）表达的意义，采用天狼猩红染色观察心肌间质纤维化，免疫组织化学检测心肌组织和培养心肌细胞TGFβ1表达。 - Conclusion Senile systolic hypertension left ventricular hypertrophy is closely related to heart failure, it is helpful to delay the occurrence of heart failure to improve left ventricular remodel.
结论老年收缩期高血压左室肥厚与心衰密切相关，积极改善左心室重塑有助于延缓心衰的发生。 - Cardiac hypertrophy is an accommodation response to almost all of factors resulted in heart diseases, but long-term cardiac hypertrophy will lead to heart ischemia, heart failure, and sudden death, which is one of the most important cause for cardiovascular diseases death.
心肌肥厚为心脏对几乎所有心脏病变因素的一种适应性反应，但长期的心肌肥厚最终将导致失代偿，而发生心肌缺血、心力衰竭和猝死，是心血管病患者死亡的重要原因之一。 - Myocardial hypertrophy is an important pathological character of congestive heart failure. Myocardial hypertrophy badly harms the patients 'health and lowers their life quality.
心肌肥大是慢性心力衰竭的重要病理特征，严重危害患者健康、降低生活质量。 - Method The animal models of renal hypertension rats was made by two kidney one clip as experiment group, then, the blood pressure and cardiac hypertrophy parameter of rats and levels of PDGF β receptor in heart were measured at 4 week and 8 week respectively.
方法以二肾一夹（2K1C）高血压大鼠动物模型为实验组，观察术后4、8周动脉血压、心肌肥大指数及心肌组织PDGF-β受体含量的变化，并与对照组进行比较。 - Objective: Cardiac hypertrophy is one of adaptive responses of the heart to a variety of pathological stimuli, including hypertension, myocardial infarction, congenital heart disease, heart valvula disease, and perturbations in sarcomeric function, and is the main reason of the refractory heart failure.
目的：心肌肥大是高血压、心脏瓣膜病、急性心肌梗塞及先天性心脏病等负荷性心脏病的并发症，是心肌细胞对多种病理刺激的一种适应性反应，也是导致顽固性心衰的主要原因。 - Background and aim of the study: With the development of hypertension, left ventricular hypertrophy is the foremost characteristic at the aspect of heart damage, which leads to heart failure ultimately.
研究背景和目的：高血压病进展时，对心脏的损害主要表现为左心室肥厚，最终可导致心衰的发生。 - Objective: Cardiomyocyte hypertrophy is not only a significant predictor of sudden death of cardiovascular diseases, but also an important risk factor of heart failure. As a result, it is of great importance to study the mechanisms of cardiac hypertrophy.
目的：心肌肥大不仅是心血管疾病发生猝死的重要预测因子，还是导致心力衰竭的危险因素，因而研究心肌肥大的发生机制对于心力衰竭和其它肥厚型心肌病变的防治具有重要意义。 - In view of AMPK in cardiac hypertrophy and fibrosis, we hypothesized that activation of AMPK by berberine may inhibit the cardiac hypertrophy and fibrosis development. AMPK α 2 is the main catalytic subunit in the heart, where it is predominantly expressed in cardiac myocytes.
大量的机制研究发现，其主要是通过激活AMPK而起作用。鉴于AMPK在心肌肥厚及纤维化中的作用，我们推测小檗碱可能通过激活AMPK抑制心肌肥厚及纤维化的发生发展。 - The earliest clinical appears features are left ventricular diastolic dysfunction, decreased left ventricular compliance and left ventricular hypertrophy. Then it develops to contractive function dysfunction, Left ventricular expanding and showing all score of cardiac arrhythmia, finally leading to the heart failure.
临床最早出现左心室舒张功能障碍，并左心室顺应性降低和左心室肥厚；然后发展为收缩功能障碍，左心室扩张并出现各种心律失常以致心力衰竭。 - Although pathological hypertrophy of the myocardium is temporarily compensatory, prolonged hypertrophy may result in ventricular arrhythmias, heart failure, and subsequent cardiovascular mortality.
虽然心肌细胞最初的生理性肥大是一种代偿机制，但是持续长久的心肌肥厚可能导致心律失常、心力衰竭甚至猝死。 - Cardiac hypertrophy is an adaptive response of the heart to pressure overload, prolonged cardiac hypertrophy may result in cardiac dysfunction leading to subsequent cardio-vascular events, such as congestive heart failure and sudden death.
心肌肥厚是心脏对压力负荷的一种代偿性反应，长时间心肌肥厚将导致心脏功能紊乱从而促使心血管事件如心力衰竭和猝死的发生。 - It is now well accepted that cardiac hypertrophy belongs to adaptive responses of the heart to various stimuli and is closely related to pressure overload and abnormal neuroendocrine.
目前认为心肌肥大是心脏对多种病理刺激的一种适应性反应，其形成与压力负荷增加、神经内分泌系统功能异常密切相关。